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Accumulating evidence has indicated that the activation of the PI3K/Akt pathway involves the improvement of neurological functional recovery on ICH (Niu and Hu, 2017

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271 Feeding endothelial cell-specific inactivated mice a high-calorie diet improved insulin resistance and decreased fat accumulation, 271 suggesting that the suppression of endothelial p53 could be a new target for the treatment of obese patients with metabolic abnormalities

To test whether Humanin could protect against lipotoxicity-induced cell death, hepatocytes were treated with palmitate in the presence or absence of Humanin

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